A. Revhaug (auth.), Professor Arthur Revhaug (eds.)'s Acute Catabolic State PDF

By A. Revhaug (auth.), Professor Arthur Revhaug (eds.)

ISBN-10: 3642488013

ISBN-13: 9783642488016

ISBN-10: 364248803X

ISBN-13: 9783642488030

This objective of this ebook is to target a really universal state of affairs noticeable in clinical perform, the intense catabolic nation. This pathophysiological take a seat­ uation isn't mentioned as a separate entity, most likely since it is noticeable in all specialties of medication and effects from a very good variety of brokers, insults, and ailments. It hence excessive time to collect an important and up to date wisdom in this entity, and the first objective of the ebook is to supply a set of up to date info at the acute catabolic nation. one other goal of the publication is to clarify that, except the final reaction of the total organism through the acute catabolic country, a chain of organ-specific responses also will occur, which also needs to be thought of in the course of therapy. It has develop into very away from those organ-spe­ from engaged on this ebook that present wisdom cific responses is especially sparse and in a few parts virtually nonexistent; this ebook hence additionally makes a speciality of the responses and alterations which occur in numerous organs in the course of the acute catabolic nation and the inter­ motion among those organs and their responses.

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Glucagon, catecholamines, insulin, and growth hormone, has been well established [1], but for over 100 years neurochemical and physiological evidence supporting the role of the brain as the integrative station for neural and hormonal regulation of peripheral metabolism has been reported by several studies [2-9]. Primarily, the hypothalamus is considered as one of the main structures involved in integrating afferent and efferent signals [2, 10], and their activities appear to be modulated by the continuous fluctuations in plasma glucose concentrations [7].

The influence of anterior pituitary extract on the metabolic response of the rat to injury. J EndocrinoI2:468-474 4. Benedict FG (1915) A study of prolonged fasting. Carnegie Institute of Washington, publication 203 5. Cahill GF (1970) Starvation in man. N Engl J Med 12:668:-675 6. Fenn WO, Haege LF (1940) The deposition of glycogen in water in the livers of cats. J Bioi Chern 136:87-101 7. Cahill GF, Herrera MG, Morgan AP et al. (1966) Hormone-fuel interrelationships during fasting. J Clin Invest 45: 1751-1769 8.

Am J Physiol 258:E288-E296 38. Molina PE, Jabbour K, Williams PE, Abumrad NN (1994) Ethanol modulates hepatic glucose production during insulin-induced hypoglycemia by inhibiting net hepatic lactate uptake. Am J Physiol (in press) 39. Iguchi A, Kunoh Y, Miura H et al. (1989) Central nervous system control of glycogenolysis and gluconeogenesis in fed and fasted rat liver. Metabolism 38:1216-1221 40. Matsunaga H, Iguchi A, Yatomi A et al. (1989) The relative importance of nervous system and hormones to the 2-deoxy-D-glucose-induced hyperglycemia in fed rats.

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Acute Catabolic State by A. Revhaug (auth.), Professor Arthur Revhaug (eds.)


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